Medically reviewed by Dr Sin Yong · Last reviewed · 9 min read · Assessed personally by Dr Sin Yong · Jump to questions
Melasma is a chronic, relapsing pigment disorder — symmetrical soft-edged brown patches across the cheeks, forehead or upper lip. It is not simple sun damage: hormones, visible light, blood vessels and dermal inflammation all feed it, which is why it behaves unlike any other brown patch and why the honest goal is control, not cure.
WhatsApp Dr Sin Yong →Melasma is a chronic, relapsing pigment disorder that causes symmetrical, soft-edged brown patches on the cheeks, forehead or upper lip. Hormones, visible light, blood vessels and dermal inflammation all contribute, so it behaves unlike ordinary sun damage. Diagnosis comes first, and management aims at long-term control rather than a one-off cure.
Also called: chloasma, mask of pregnancy, 黄褐斑, 肝斑
LookalikesOften confused with Hori's naevus and sun spots: melasma is a symmetrical, soft-edged brown patch that darkens with sun and heat, Hori's naevus is speckled blue-grey macules over the cheekbones, and a sun spot is a discrete, sharp-edged spot. Hori's naevus (ABNOM).


Current pathogenesis reviews describe melasma as far more than a melanin problem: genetically susceptible skin under chronic light exposure develops overactive melanocytes, but also an altered basement membrane, increased vascularity and low-grade dermal inflammation — the whole neighbourhood participates, not just the pigment cells [1]. This is why melasma relapses after treatments that would durably clear a sunspot, and why heat and aggressive lasers can make it rebound darker. Hormones layer on top: pregnancy and hormonal medication are classic triggers, which is also why melasma so often begins in a woman's 30s.
You cannot choose your genetics or undo a pregnancy trigger. You can control light — and the evidence here is specific: visible light, not only UV, drives melasma pigmentation, which means window glass and indoor exposure count, and standard clear sunscreens are incomplete protection. Tinted sunscreens containing iron oxides block the visible wavelengths implicated [1,3]. Heat is the other underrated trigger — saunas, steam rooms, aggressive treatments. A melasma plan that ignores light and heat management is a plan built to fail.

Melasma is described in two ways at once: by where the patches sit on the face, and by how deep the pigment lies in the skin. Both matter, because together they shape how quickly a plan can move and which steps are worth taking. Neither can be judged reliably from a selfie, which is why the type is named at examination before any treatment is discussed.

The facial pattern is the part you can see in the mirror. A centrofacial pattern spreads across the forehead, cheeks, nose, upper lip and chin. A malar pattern sits on the cheeks and nose. A mandibular pattern runs along the jawline. Some people also have patches on the forearms, which follow the same biology. The pattern helps to separate melasma from its lookalikes: a symmetrical, soft-edged patch behaves like melasma, while discrete sharp-edged spots suggest sun spots, and speckled blue-grey dots over the cheekbones suggest Hori's naevus.
Depth is the part you cannot see. Under a Wood's lamp and the dermatoscope, pigment in the epidermis shows up with sharper contrast, pigment in the dermis stays blurred and grey-brown, and many faces show a mixed picture. Dermal pigment is held by cells deeper in the skin and clears more slowly, so the honest pace of a plan follows the depth.
| Type | What it looks like | How it is checked | What it means for the plan |
|---|---|---|---|
| Centrofacial pattern | Patches across forehead, cheeks, nose, upper lip and chin | Examination and photographs in standard light | A wider area to protect and treat; trigger control matters more |
| Malar pattern | Patches on the cheeks and nose | Examination; Hori's naevus excluded | Often a more limited area; mimics are common here |
| Mandibular pattern | Patches along the jawline | Examination; post-inflammatory marks excluded | Friction and irritation are reviewed as triggers |
| Epidermal depth | Brown, sharper contrast under the lamp | Wood's lamp and dermoscopy | Topicals and photoprotection tend to show progress sooner |
| Dermal or mixed depth | Grey-brown, blurred edges | Wood's lamp and dermoscopy | A slower, more conservative plan; heat is avoided |
The type is not fixed for life. Patches can spread in pregnancy, lighten with strict protection and return with sun, heat or hormonal change. That is why the plan is reviewed rather than set once, and why the melasma treatment page sets out the option matrix by depth in detail.
The network meta-analysis across melasma treatments ranks combination approaches highest: photoprotection plus evidence-based topicals (with triple-combination creams and related regimens leading), with oral and procedural options — including conservative low-fluence laser toning — as adjuncts rather than first weapons [2,3]. Aggressive monotherapy lasers rank poorly precisely because rebound is a documented outcome. Dr Sin Yong's staged protocol on the melasma treatment page follows this hierarchy: diagnose first (see which brown is yours — melasma coexisting with other pigment types is common), stabilise with topicals and photoprotection, then add gentle energy-based work such as laser toning where indicated, with maintenance planned from day one.
One strong laser to “remove it” — melasma punishes heat and haste with rebound pigmentation. Bleaching creams from unregulated sources — some contain unlisted steroids or high-dose hydroquinone that damage skin with long-term unsupervised use. Scrubbing — friction is itself a melasma trigger. And any clinic promising a cure — the literature is unambiguous that melasma is a chronic relapsing condition; a promise of cure is a promise of your disappointment.
“Melasma punishes aggression — the laser that clears a sunspot in one session can hand a melasma patient a darker patch than they started with.”
— Dr Sin Yong
Melasma is treated by a doctor in stages: diagnosis first, then control of the triggers, then topical and oral medicine where suitable, and gentle energy work once the skin is stable, not before. The order matters more than the device. Starting with a strong laser on a face that has not been diagnosed or protected is the commonest route to rebound darkening, and the reason so many people arrive having tried treatment elsewhere.
Singapore adds two pressures. Most skin here is Fitzpatrick III to V, which forms pigment readily after heat or inflammation, and the ultraviolet index stays high all year and the heat adds to it. A plan that ignores daily light and heat exposure does not hold, whatever is done in clinic.
| Step | What happens | Why it comes at this point |
|---|---|---|
| 1. Diagnosis | Examination with Wood's lamp and dermoscopy; Hori's naevus, sun spots and post-inflammatory marks separated; hormonal medication and pregnancy plans reviewed | Each brown has a different plan, and melasma often coexists with another type |
| 2. Light and heat control | Daily tinted sunscreen with iron oxides for visible light, shade and a hat, and fewer heat triggers such as saunas | Without this, every later step is undone |
| 3. Topical loading | Prescription and skincare agents chosen for the skin, such as hydroquinone in planned courses, tretinoin, azelaic acid or tranexamic acid | Settles pigment production before any energy is considered |
| 4. Oral medicine where suitable | Oral tranexamic acid, considered after clotting risk, smoking and hormonal contraception are reviewed | A prescription option for some; not used where the clotting risk is raised or in pregnancy |
| 5. Gentle energy, if indicated | Conservative work starting with R2 Glow alone, or no laser at all, within the R2T2 protocol | Addresses the inflammatory drive without the heat that triggers rebound |
| 6. Maintenance and review | Photoprotection continues; topicals are stepped down; relapse is planned for | Melasma is controlled rather than cured |
Dr Sin Yong, an aesthetic physician, assesses and treats every patient personally on Orchard Road. Where a patch is changing, raised or unlike the rest, it is examined first and referred for dermatological assessment rather than treated as melasma. Pregnancy and breastfeeding change the plan most: elective laser is deferred and most prescription agents are avoided, so photoprotection becomes the main measure until the plan can resume.
Melasma is managed mostly at home, day to day, with a doctor setting the plan and stepping in where home care cannot reach. What you do every morning matters more than any single clinic visit, because light, heat and friction keep switching the pigment cells back on.
Prescription-strength hydroquinone, tretinoin and oral tranexamic acid need a doctor to decide the strength, how long a course runs and who should avoid them, for example in pregnancy or where the clotting risk is raised. Lightening creams from salons or online sellers may contain undeclared steroids or unsafe hydroquinone. Home IPL and laser gadgets add heat to a heat-sensitive condition.
Most skin in Singapore is Fitzpatrick type III to V, which makes pigment readily after irritation. A routine that stings or peels can leave post-inflammatory hyperpigmentation on top of the melasma, and the two then need separating before anything else is changed. Start one new product at a time and stop anything that burns.
See a doctor when patches keep spreading despite a daily tinted sunscreen, when a cream has left the skin red, thin or with visible vessels, or when one spot looks different from the rest. The assessment and staged plan are set out on melasma treatment in Singapore.
Melasma has humbled every aggressive laser ever pointed at it, and it will humble the next one too. I manage it the way the evidence says: light control, topicals, patience, and gentle energy in its supporting role — with maintenance planned from day one. Any clinic promising a melasma cure is promising your relapse.
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| Type / look-alike | How to recognise it | What it needs | What does not work |
|---|---|---|---|
| Melasma | Mirror-image, soft-edged patches on cheeks, forehead or upper lip | Diagnosis, tinted photoprotection and topicals; gentle energy as adjunct | One strong laser, which risks rebound pigmentation |
| Melasma with other pigment | Melasma patches mixed with spots or marks | Each component identified, then a staged plan | Treating every brown patch the same way |
| Sun spots (solar lentigines) | Discrete, sharply bordered spots on sun-exposed skin | Diagnosis, then pigment-targeted treatment | Whitening facials and scrubbing |
| Post-inflammatory hyperpigmentation | Flat brown-grey mark where acne or injury occurred | Control the inflammation, protect from sun, then treat pigment | Scrubbing and picking, which prolong inflammation |
| Freckles | Small, genetic, UV-responsive spots that darken with sun | Treatment only if bothersome, with sun protection | Expecting them to stay away without sun protection |
Heat and aggressive settings can make melasma rebound darker than before.
Visible light also drives melasma, so tinted sunscreens containing iron oxides, which block those wavelengths, are advised.
Because the disease is still there — overactive melanocytes, vascular changes and light sensitivity persist under cleared skin. Clearing is a state to be maintained, not a cure that ends care.
Pregnancy is a classic trigger — the hormonal surge activates susceptible pigment cells. Some post-pregnancy melasma fades; patches that persist months after delivery usually need active management.
Yes — visible light, including from bright indoor environments and through windows, measurably stimulates melasma pigmentation. This is why iron-oxide tinted sunscreen is standard melasma advice.
Gentle, low-energy laser protocols have a supporting role within a combination plan. High-energy 'removal' attempts are the classic way melasma gets worse — device, settings and sequence matter more than the word 'laser'.
Genetic susceptibility plus your particular mix of hormones and light exposure. Fitzpatrick III–V skin — most Singaporean skin — carries the highest risk.
Photoprotection stops the fuelling but rarely erases established pigment alone. It is the foundation every other treatment stands on — necessary, seldom sufficient.
Patient guide (PDF): Melasma: daily management — a printable summary of this page, medically reviewed by Dr Sin Yong. General information, not a substitute for assessment.
Medically reviewed by Dr Sin Yong, MBBS (NUS), MRCS (Edinburgh) · Last updated 21 October 2026 · Editorial policy
Every treatment is performed by Dr Sin Yong personally. Singapore Medical Council registered · invited to share his clinical expertise internationally as a Key Opinion Leader for CLASSYS, DEKA, HIRONIC, FOTONA, ALMA, LUTRONIC and ILOODA · HSA-registered devices · ultrasound-guided correction
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