Medically reviewed by Dr Sin Yong · Last reviewed · 9 min read · Assessed personally by Dr Sin Yong · Jump to questions
This page covers marks left by inflammation. For the other types of hyperpigmentation, see hyperpigmentation types on the face; for treatment across all types, see pigmentation treatment in Singapore.
PIH is the footprint inflammation leaves in pigmented skin: a flat brown, grey or slate mark exactly where a pimple, bite, rash or overly aggressive treatment used to be. The insult is gone; the pigment it provoked remains. In Fitzpatrick III–V skin — most of Singapore — PIH routinely outlasts and outbothers the problem that caused it.
WhatsApp Dr Sin Yong →Post-inflammatory hyperpigmentation (PIH) is a flat brown, grey or slate mark left where inflammation from acne, a bite, a rash or an over-aggressive treatment stimulated pigment production. It is common in Fitzpatrick III–V skin. Depth decides the course: brown epidermal marks fade sooner than grey dermal ones, so treatment starts with stopping the inflammation and strict photoprotection.
Also called: PIH, acne dark marks, 痘印, 炎症后色素沉着
LookalikesOften confused with post-inflammatory erythema: PIH is a flat brown, grey or slate mark that keeps its colour under a pressed glass slide, while PIE is a flat red or pink mark that blanches under the same pressure. red acne marks (PIE).
PIH is the footprint inflammation leaves in pigmented skin: cytokines released during a skin insult stimulate melanocytes, and in darker skin the response is brisk. Pigment that stays in the epidermis reads brown and fades sooner; pigment that drops through a disrupted basement membrane reads grey-slate and is held in the dermis. Dr Sin Yong assesses the depth, stops the cause, insists on photoprotection, then adds topicals and conservative laser.
Post-inflammatory hyperpigmentation (PIH) is a flat brown, grey or slate mark left where a pimple, rash, bite or injury inflamed the skin. PIH treatment in Singapore follows an order: calm the inflammation, protect from UV and visible light, add lightening topicals under supervision, and consider low-fluence laser last. Dr Sin Yong assesses the depth of the pigment first.


Inflammation is a megaphone aimed at melanocytes: cytokines released during any skin insult stimulate pigment production, and in darker skin types the response is brisk and generous. When the pigment stays in the epidermis, the mark is brown and fades as skin turns over. When inflammation disrupts the basement membrane, pigment drops into the dermis, where immune cells hold it — those marks read grey-slate and can sit for years [1]. Depth, not darkness, is what determines how long you will be looking at it — and depth is assessable in clinic.
Three different leftovers get called “acne marks”. A flat brown-grey mark is PIH — pigment. A flat pink-red mark that blanches under a pressed glass is post-inflammatory erythema — vessels, not pigment, treated on vascular principles like thread veins. A dent or dip you can feel is a true acne scar — a structural problem needing structural treatment. Mixing these up wastes months: pigment treatment does nothing for redness, and neither does anything for a dent. One examination sorts all three — and where active acne is still seeding new marks, the acne itself gets treated first, or the tail never ends.

Post-inflammatory hyperpigmentation on the body follows the same rule as on the face: inflammation came first, pigment followed. On the body the triggers are different, the skin renews more slowly, and the marks are easier to mistake for other conditions, so they are worth understanding on their own.
Stop the trigger first: treat bites and eczema early so they are not scratched, wear loose, breathable clothing, and avoid harsh scrubs, loofahs and lemon juice. Use sunscreen on legs and arms that are exposed. What you should not do is buy body whitening creams of unknown content, because some contain undeclared strong agents; hydroquinone is a prescription medicine in Singapore. Grey-slate marks that sit deep in the dermis do not respond to anything applied at home.
Fitzpatrick III to V skin answers inflammation with more pigment, and marks on the lower legs often last longer than marks on the face. Laser on the body is therefore used conservatively and only after the trigger is controlled.
See a doctor if the skin is velvety or thickened, a patch keeps returning in the same spot, patches are scaly or spreading, itching continues, or marks appeared after a laser or peel. Back and chest marks are covered under back acne treatment, and the pigment laser approach under pico laser for PIH.
The systematic reviews converge on a sequence [1,2]. First, stop the inflammation — active acne, eczema or picking makes PIH a moving target. Second, strict photoprotection: UV and visible light deepen existing marks, so tinted sunscreen is baseline treatment, not an accessory. Third, evidence-based lightening topicals — retinoids, azelaic acid, hydroquinone-class and newer agents under supervision. Fourth, where topicals plateau, conservative laser work: low-fluence pigment protocols such as laser toning and picosecond systems on the T2 platform — with the reviews of laser treatment in skin of colour emphasising restraint, because the same devices set aggressively cause the condition they treat [2]. The overlapping differential with other browns is covered in which brown is yours.
PIH treatment in Singapore goes wrong most often through sequence, not through choice of product. The mark is the footprint of inflammation, so anything that inflames the skin again, from scrubbing to an aggressive laser, can add a mark rather than remove one. The four stages below are the order Dr Sin Yong works in, and each stage is only moved past when the one before it is in place.
The laser is the last step in PIH treatment, not the first, because the same device set aggressively causes the mark it is meant to clear.— Dr Sin Yong

| Stage | What is done | Why it comes here | Read more |
|---|---|---|---|
| 1. Barrier first | Stop the trigger: active acne is controlled medically, eczema is settled, picking stops, and scrubs and DIY acids are dropped | While inflammation continues, PIH is a moving target and every new lesion can leave another mark | Acne treatment |
| 2. Photoprotection | Daily broad-spectrum sunscreen, tinted where possible, with shade and hats | UV and visible light deepen existing marks, so protection is treatment, not an accessory | Which brown is yours |
| 3. Supervised topicals | Retinoids, azelaic acid, hydroquinone-class agents and newer lightening agents, prescribed and reviewed | They lighten epidermal pigment and suppress new pigment without adding inflammation when used correctly | Pigmentation guide |
| 4. Low-fluence laser | Where topicals plateau and the depth has been assessed: laser toning or picosecond settings on the T2 platform, conservative and reviewed | Last because the same devices set aggressively cause the condition they treat, particularly in darker skin | Pico laser for PIH |
Depth decides how far down the list a mark needs to go. Brown, sharper-edged epidermal PIH often clears with the first three stages. Grey-slate dermal PIH, where pigment has dropped through the basement membrane, is the one more likely to need conservative laser work, and it is also the one most easily made worse. That is why the assessment separates the two before any device is switched on, and why post-inflammatory erythema, which is red and blanches under pressed glass, is sent down a vascular route instead.
Darkening after a peel or laser elsewhere is treated as the same problem: the cause is reviewed, the barrier is restored, and the sequence starts again from stage one rather than repeating the treatment that caused it. The CO2 laser in Asian skin page and laser toning explain how energy settings are kept conservative in pigment-prone skin.
Scrubbing the marks — friction is inflammation, and inflammation is the disease. Lemon juice and DIY acids — chemical irritation on pigment-prone skin is a PIH recipe. Bleaching creams from unregulated sources — unsupervised strong agents can leave lasting damage including paradoxical darkening. Picking the next pimple — every squeezed lesion is a PIH order form. And impatience: dermal pigment clears on biology's schedule, and any clinic promising erasure in a fortnight is selling the calendar, not the outcome.
“In Singapore skin, the pimple is rarely the problem — the three-month brown souvenir it leaves is, and every squeeze places another order.”
— Dr Sin Yong
Laser has a place in post-inflammatory hyperpigmentation only after the inflammation has been controlled and topical treatment has been tried, because any heat or injury can add a new mark to skin that already darkens easily.
Who should wait: anyone with active acne, eczema or other inflammation in the area; a recent tan; pregnancy or breastfeeding; photosensitising medicines; recent isotretinoin, where the doctor advises on timing; a history of keloid scarring; and anyone whose dark marks have not yet been diagnosed, since melasma, Hori’s naevus and pigmented lesions can look similar.
Common and expected: brief redness and warmth; treated pigment can look darker before it flakes. The risks that matter: rebound darkening if the energy is too high or treatments are too close together; pale or mottled spots after frequent repeated toning; and, rarely, blistering or crusting. These risks are higher in darker skin and after sun exposure, which is why settings are conservative, intervals are set at review, and strict sun protection continues throughout.
Before any laser, tell the doctor about all creams and medicines, recent procedures and sun exposure. The pico approach to PIH is on pico laser for PIH.
In our skin, the pimple is rarely the real enemy — the three-month brown souvenir is. I spend more consult time on PIH than on the acne that caused it, and my first prescription is always the same: stop the picking, start the photoprotection, and let restraint do what aggression cannot. Every squeeze is an order form for another mark.
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| Type / look-alike | How to recognise it | What it needs | What does not work |
|---|---|---|---|
| Epidermal PIH | Brown, sharper-edged mark with pigment sitting high | Stop inflammation, protect from sun, lightening topicals | Scrubbing, lemon juice and DIY acids |
| Dermal PIH | Grey-slate mark with pigment dropped deep | Depth assessment, then topicals and conservative laser where indicated | Expecting a quick fade or aggressive laser |
| Post-inflammatory erythema | Flat pink-red mark that blanches under pressed glass | Treated on vascular principles, not pigment treatment | Pigment lightening creams will not help |
| Acne scar | A dent or dip you can feel | Structural treatment of the dent itself | Pigment treatment, which does not address texture |
| Treatment-induced PIH | Darkening after a peel or laser | Cause reviewed, then the same stepwise approach | Repeating the aggressive treatment |
Depth, not darkness, decides the course, and depth is assessed in clinic.
Unsupervised strong agents and irritation can add inflammation or cause paradoxical darkening.
Epidermal (brown) marks typically fade over several months. Dermal (grey-slate) pigment can persist for years without treatment. Depth decides the timeline, and assessment reads the depth.
Melanocyte reactivity scales with constitutive pigment — Fitzpatrick III–V skin answers inflammation with more melanin, faster. It is biology, not fragility.
No — PIH is flat pigment; a scar is a texture change you can feel. They often coexist after acne, and each needs its own treatment plan.
Conservative, low-energy pigment protocols have supporting evidence — but aggressive settings cause PIH, so device restraint is the entire art in darker skin. This is a physician's-judgement treatment.
Yes — UV and visible light actively deepen existing PIH. Strict photoprotection is the difference between a mark that fades and one that entrenches.
Treatment-induced PIH — the inflammation from an aggressive procedure triggered the same pathway as a pimple. It usually fades, and it is precisely why settings are chosen conservatively for your skin type here.
PIH, post-inflammatory hyperpigmentation, is a flat brown, grey or slate mark left where inflammation from acne, a bite, a rash or an over-aggressive treatment stimulated pigment production. Depth decides the course: brown epidermal marks fade over months as skin turns over, while grey-slate pigment that has dropped into the dermis can persist for years. It is common in Fitzpatrick III to V skin. Treatment starts with stopping the inflammation and strict photoprotection; see PIH.
Medically reviewed by Dr Sin Yong, MBBS (NUS), MRCS (Edinburgh) · Last updated 5 November 2026 · Editorial policy
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