Medically reviewed by Dr Sin Yong · Last reviewed · 13 min read · Doctor-performed, never delegated · Jump to questions
Dr Sin Yong · MBBS (NUS) · MRCS (Edin) · MSc Aesthetic Medicine with Distinction (Queen Mary, London) · MSc Practical Dermatology (Cardiff) · International KOL
The two commonest pigmentation problems in Singapore look alike — and are treated almost oppositely. Getting the diagnosis right matters more than the technology.
Melasma treatment in Singapore starts with diagnosis, because melasma is a hormonal, chronic, relapsing and heat-sensitive pigmentation that is managed very differently from sun spots (solar lentigines). Dr Sin Yong separates the two at assessment before choosing any device, since an approach suited to one can aggravate the other.
Also called: chloasma, mask of pregnancy, 黄褐斑, 肝斑
Patients often arrive having already had laser for what they were told was pigmentation. Whether that was the right call depends entirely on which pigmentation it was.
Sun spots — solar lentigines — are deposits. They sit where UV has accumulated over years, they have defined borders, and they behave relatively predictably.
Melasma is a different condition altogether. It is hormonal, chronic and relapsing, it is driven by melanocytes that are overactive rather than more numerous, and it is heat-sensitive. It distributes in the centrofacial, malar and mandibular patterns rather than as discrete spots.
The two can coexist on the same face, which is precisely why the assessment separates them before any device is chosen. Other pigmentation types and post-inflammatory hyperpigmentation are separated at the same time.
Singapore sits roughly one degree north of the equator. There is no seasonal low in UV, so the exposure is year-round rather than summer-weighted — which is one reason melasma behaves differently here than it does in temperate advice written for temperate climates.
The less familiar part is heat. Infrared and visible light are triggers in their own right, independent of UV. That means a hot yoga class, a steamy walk to the MRT, a kitchen, or a sunscreen that covers UVB and UVA but not visible light can all contribute — and so can a heat-generating laser applied to the wrong diagnosis.
Practical management therefore has to address heat exposure and daily photoprotection alongside whatever is done in clinic. Treatment that ignores the triggers is treating one half of the problem.
“The two commonest pigmentation problems in Singapore look alike and are treated almost oppositely.”
Dr Sin YongOn melasma and sun spots · from his Instagram explainer series

With a sun spot, more aggressive settings often shorten the route. With melasma the same instinct works against you, because thermal injury is itself one of the triggers.
Pushed too hard, melasma commonly rebounds — returning darker than the starting point, and harder to manage than it was before. That risk is higher in Fitzpatrick III–V skin, which describes the majority of patients in Singapore.
The approach here is deliberately conservative and staged, with settings chosen for the phototype in front of me rather than for a protocol written elsewhere. Where a device is appropriate at all, it is one part of a plan that also covers triggers, photoprotection and topical management. Response varies between individuals and is reviewed in person.
“Melasma is managed, not cured. A clinic promising a cure is promising a relapse.”
Dr Sin YongOn pigmentation assessment
Melasma is planned individually, so cost follows the plan rather than a fixed list. Four things shape it:
A figure quoted before assessment would not reflect your presentation. Cost is set out clearly at consultation, before anything is agreed.
Melasma treatment covers creams, tablets and in-clinic work, and the cautions differ for each. Pregnancy and breastfeeding change the plan most: melasma often appears or worsens then, elective laser is generally deferred, and prescription agents such as hydroquinone, tretinoin and oral tranexamic acid are usually avoided, so gentle photoprotection becomes the main measure.
Oral tranexamic acid affects clotting, so a personal or family history of blood clots, a known clotting disorder, smoking and the use of combined hormonal contraception are reviewed before it is considered, and it is not used where the clotting risk is raised.
Laser or toning is deferred with a recent tan, active dermatitis or irritation from strong actives, or photosensitising medication, and it is not started while the diagnosis is uncertain. A patch that is changing, raised or unlike the rest is examined first and referred for dermatological assessment where needed. Suitability is decided at consultation.
The main risk in melasma treatment is making the pigment worse. Heat-based or aggressive laser can trigger rebound darkening, and frequent repeated low-energy toning of the same area has been linked to pale, mottled spots that can be slow to recover. That is why in-clinic work here is conservative, staged and only one part of the plan.
Creams carry their own risks: irritation, redness and peeling from tretinoin or hydroquinone and, with prolonged unsupervised hydroquinone use, a bluish-grey darkening called exogenous ochronosis. Oral tranexamic acid can cause stomach upset or lighter periods and, rarely, a blood clot. In a retrospective study from Singapore's National Skin Centre of 561 patients given oral tranexamic acid for melasma, 7.1% had adverse effects, mostly transient, and one developed a deep vein thrombosis and was later found to have an inherited clotting disorder.
Risks are reduced by confirming the diagnosis first, screening for clotting risk, using prescription agents in supervised courses, choosing settings for Fitzpatrick III to V skin and reviewing response before anything is escalated.
The sequence below describes phases, not a timetable: how quickly each passes depends on the settings used, the area and the person, and is discussed at consultation.
People hoping for a one-off clearance, or unable to commit to daily photoprotection and heat avoidance, tend to respond poorly, because melasma is chronic and relapsing.
The fee follows the plan, not a fixed list. It depends on what the assessment finds, since epidermal, dermal and mixed patterns are managed differently, and on the area involved, from a limited malar patch to a centrofacial distribution. It also reflects whether topical care, trigger control and in-clinic work are combined, and how the plan is staged. A written quote is given at consultation, after assessment, before anything is agreed. The consultation also decides whether treatment is advised at all.
How quotes work at this practice: how we quote.
Heat is itself a trigger, so higher energy can darken melasma rather than lighten it.
They look alike but behave differently, and an approach suited to one can aggravate the other.
Brown marks on the face look alike from a distance, and the wrong label leads to the wrong treatment. Melasma, solar lentigines (sun spots), post-inflammatory hyperpigmentation (PIH), freckles and Hori's naevus differ in pattern, in the depth of the pigment, in what triggers them and in what makes them worse. Several can sit on the same face, which is why the examination separates them before any device or cream is chosen. Depth matters most: pigment in the epidermis, the dermis or both responds to different approaches. The table is a reading aid for understanding the examination, not a way to diagnose yourself, and a changing, raised or unusual patch is checked first.
| Brown mark | Pattern | Depth | Trigger | What helps | What worsens |
|---|---|---|---|---|---|
| Melasma | Symmetric patches on cheeks, forehead, upper lip or jaw | Epidermal, dermal or mixed | UV, visible light, heat, hormones | Trigger control, daily photoprotection, topical care, cautious staged clinic work | Heat, aggressive laser, repeated toning, unprotected sun |
| Solar lentigo (sun spot) | Discrete spots with defined borders on sun-exposed skin | Epidermal | Accumulated UV over years | Sun protection; pigment-targeting laser once diagnosed | Continued unprotected sun exposure |
| PIH | Follows the shape of earlier acne, injury or laser | Epidermal or dermal | Inflammation, friction, injury | Settling the cause, gentle care, sun protection | Picking, irritation and over-aggressive treatment |
| Freckles | Small tan spots across nose and cheeks, often familial | Epidermal | UV darkens them | Daily sun protection; laser if wanted, once confirmed | Unprotected sun exposure |
| Hori's naevus | Blue-grey to grey-brown dots over the cheekbones, both sides | Dermal | Not clearly sun- or heat-driven | Laser chosen for dermal pigment after examination | Surface-directed treatment, which does not reach dermal pigment |
Melasma is driven by melanocytes that are overactive rather than more numerous, and they respond to energy as a threat. Heat and infrared are triggers in their own right, so a device that delivers heat to the face can stimulate the very cells it is aimed at. Inflammation adds a second route: a laser that irritates the skin can leave post-inflammatory pigment on top of the melasma, and in Fitzpatrick III to V skin that overlay is more likely. The result can be a patch that returns darker than it began, and harder to manage than before. Repeated low-energy toning of the same area has also been linked to pale, mottled spots that recover slowly.
The plan guards against these routes in sequence. The diagnosis and the depth are established first, because a laser suited to a sun spot can aggravate melasma. Triggers, daily photoprotection and topical management come before any device, and in-clinic work, where it is appropriate at all, is conservative, staged and chosen for your phototype. Each step is reviewed for rebound, irritation or pale patches before anything further is done. A flare or darkening after any step is read as a signal to reassess the plan, not to repeat it with more energy, which is the instinct that most often makes melasma worse.
Melasma is chronic and relapsing, so the honest answer is that it can be managed but not reliably cleared for good. Pigment can lighten gradually under a plan built on trigger control, photoprotection and cautious treatment, but the tendency itself does not go away. Hormonal influence, heat and light stay in the background, and the same triggers that produced the first patch can produce another. Response varies between individuals and is reviewed in person, which is why no duration of improvement is promised. As Dr Sin Yong puts it, a clinic promising a cure is promising a relapse.
A realistic aim is therefore steady management rather than a one-off clearance, with a plan that does not depend on a single session. That means accepting ongoing care: daily photoprotection, heat avoidance and reviewed, staged treatment rather than a one-off intervention. It also means that a return of pigment after a hot season, a pregnancy or a change of hormonal medicine is a known pattern to be reassessed, not proof that the earlier plan failed. If melasma turns out not to be the diagnosis, as with sun spots, a different approach applies.
Maintenance is largely trigger management, and each trigger has a practical answer. Hormones are well recognised: melasma commonly appears or worsens in pregnancy and with hormonal contraception, so that history is taken at assessment, and any change to medicine is discussed with the prescriber, not made on your own. Heat is the trigger people overlook. A hot yoga class, a kitchen, a steamy walk to the MRT and heat-generating devices can all contribute, so cooling and shade matter alongside sun avoidance. Singapore sits about one degree north of the equator, so UV is high all year with no seasonal low.
Sunscreen choice matters more than most people expect. A clear sunscreen that covers UVB and UVA does not address visible light, which is also a trigger, and tinted formulations containing iron oxides are commonly used for that reason. The choice between mineral and chemical filters is largely a question of which you will wear daily and reapply through the day, and the one used consistently is the one that helps. Light passes through windows, so protection continues indoors near them. Review checks for rebound, irritation and pale patches, and a flare is treated as information that triggers have changed. Sunscreen is part of management, not a treatment on its own.
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They look alike and are treated almost oppositely. Sun spots are deposits with defined borders and respond relatively predictably to laser. Melasma is hormonal, chronic and heat-sensitive, distributes symmetrically across the centrofacial, malar or mandibular areas, and can rebound darker if treated aggressively. The two often coexist, so they are separated at assessment before any device is chosen.
Thermal energy is itself a melasma trigger, so a device chosen for the wrong diagnosis, or settings pushed too hard, can drive rebound hyperpigmentation. If pigmentation worsened after a session, the sensible step is reassessment before booking another rather than continuing the same approach.
Melasma is chronic and relapsing, so it is managed rather than removed. An honest plan starts conservatively, addresses heat and UV triggers, and includes daily photoprotection. Response varies between individuals and is assessed in person.
UV and heat. Singapore sits roughly one degree north of the equator, so UV is high year-round with no seasonal low. Heat and visible light are independent triggers, which is why hot environments, steamy commutes and heat-generating devices all matter alongside sun exposure.
Daily photoprotection is foundational, but UVB and UVA coverage alone does not address visible light, which is also a trigger. Tinted formulations containing iron oxides are commonly used for that reason. Sunscreen is part of management rather than a treatment on its own.
Hormonal influence is well recognised, which is why melasma commonly appears or worsens in pregnancy and with hormonal contraception. That context is part of the history taken at assessment, because it shapes what is realistic to expect from treatment.
Cost depends on what the assessment finds, how much of the face is involved, whether one approach or several are combined, and how the plan is staged. Because those differ substantially between patients, a figure given before assessment would not be meaningful. Cost is discussed at consultation.
Cost follows the plan rather than a price list. It depends on what assessment finds (epidermal, dermal or mixed), the area involved, whether topical care, trigger control and in-clinic work are combined, and how the plan is staged. A written quote is given at consultation, after assessment, and the consultation also decides whether treatment is advised at all.
It tends to suit people who want the pigment managed and will commit to daily photoprotection, heat avoidance and reviewed, staged care. It is a poor fit for anyone seeking a one-off clearance, or whose pigment is actually sun spots, where a different approach applies. Assessment decides which condition is present before any plan is offered.
Melasma is chronic and relapsing, so there is no fixed duration to promise. How long improvement holds depends on triggers such as UV, visible light, heat and hormones, on daily photoprotection, and on the depth of the pigment. Review timing and any maintenance are set at consultation and adjusted to how your skin responds.
It is gradual and needs ongoing care. Aggressive or heat-based laser can darken melasma, repeated toning has been linked to pale spots, and creams can irritate or, if used unsupervised for long, cause bluish-grey discolouration. Oral tranexamic acid is not suitable where clotting risk is raised. Pigment can also return after a flare of triggers.
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Medically reviewed by Dr Sin Yong, MBBS (NUS), MRCS (Edinburgh) · Last updated 7 October 2026 · Editorial policy
Patient guide (PDF): Melasma: daily management — a printable summary of this page, medically reviewed by Dr Sin Yong. General information, not a substitute for assessment.
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